Volume 15, Issue 57 (12-2024)
NCMBJ 2024, 15(57): 1-19 |
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Sabzevari Rad R. Interactions between skeletal muscles and distant organs mediated by signaling factors during exercise: a narrative review. NCMBJ 2024; 15 (57) :1-19
URL: http://ncmbjpiau.ir/article-1-1714-en.html
URL: http://ncmbjpiau.ir/article-1-1714-en.html
Department of Physical Education and Sport Sciences, Faculty of Command and management, Imam Ali Military' University, Tehran, Iran , sabzevarireza63@yahoo.com
Abstract: (169 Views)
Exercise performs an effective role withinside the prevention and remedy of numerous persistent sicknesses, which include persistent coronary heart failure, diabetes, and depression. Exercise training has been useful for such persistent sicknesses through interactions of muscle tissues and different organs. exercise results in the secretion of many myokines, which, even as affecting the signaling pathways of muscle tissues, additionally modify the features of different organs, which include the brain, fat tissue, bone, liver, and intestine. It is feasible that myokines are beneficial biomarkers for prescribing exercise to prevent and remedy diseases along with coronary heart failure, cognitive dysfunction, obesity, fatty liver, etc.
Abbreviation
BAIBA:β-aminoisobutyricacid;miRNA:MicroRNA;,DMD:Duchenne muscular dystrophy;Ace: angiotensin converting enzyme; BDNF: brain-derived neurotrophic factor; NGF: neurotrophic factor; CTSB: Cathepsin B; AMPK: 5' AMP-activated protein kinase; KAT: kynurenine aminotransferase; PRR: pattern recognition receptor; NFκB: nuclear factor kappa B; NAFLD: non-alcoholic fatty liver disease; SCFAs: Short chain fatty acids; Gpr41: G-Protein Coupled Receptor 41; T2DM: Type 2 diabetic mellitus; Akt: Protein kinase B; CLK2 content: Dual specificity protein kinase2; HSP72: heat shock protein 72; SOCS3: suppressors-of-cytokine-signaling 3; GSK3B: glycogen synthetic kinase 3B; MIF: macrophage migration inhibitory factor; CHD: coronary heart disease; TLRs: Toll-like receptors; MAMP: microbe-associated molecular pattern; PGC-1α: Peroxisome proliferator-activated receptor-gamma coactivator -1alpha; NLR: nucleotide-binding oligomerization domain (NOD)-like receptor;T-RFLP analysis: Terminal restriction fragment length polymorphism;NOD: nucleotide-binding oligomerization domain; AMPK: adenosine 5′-monophosphate-activated protein kinase; FSTL1: follistatin-like 1; GLP: glucagon-like peptide; LIF: leukemia inhibitory factor; Metrnl: meteorin-like; TGF-β: transforming growth factor β; TNF: tumor necrosis factor; UCP1: uncoupling protein 1; GLP-1: glucagon-like peptide 1; IPost: Ischemic postconditioning; c-Met: mesenchymal-epithelial transition factor; SRF: serum response factor; NYHA: The New York Heart Association; Fas: fatty acid synthase; Acc : acetyl-CoA carboxylase; Cpt-1: Carnitine palmitoyltransferase I; β-Had: beta-Hydroxyacyl-CoA dehydrogenase; OLETF: Otsuka Long-Evans Tokushima Fatty; FATP4: fatty acid transporter4; GSK3β: glycogen synthetic kinase 3 β.
Abbreviation
BAIBA:β-aminoisobutyricacid;miRNA:MicroRNA;,DMD:Duchenne muscular dystrophy;Ace: angiotensin converting enzyme; BDNF: brain-derived neurotrophic factor; NGF: neurotrophic factor; CTSB: Cathepsin B; AMPK: 5' AMP-activated protein kinase; KAT: kynurenine aminotransferase; PRR: pattern recognition receptor; NFκB: nuclear factor kappa B; NAFLD: non-alcoholic fatty liver disease; SCFAs: Short chain fatty acids; Gpr41: G-Protein Coupled Receptor 41; T2DM: Type 2 diabetic mellitus; Akt: Protein kinase B; CLK2 content: Dual specificity protein kinase2; HSP72: heat shock protein 72; SOCS3: suppressors-of-cytokine-signaling 3; GSK3B: glycogen synthetic kinase 3B; MIF: macrophage migration inhibitory factor; CHD: coronary heart disease; TLRs: Toll-like receptors; MAMP: microbe-associated molecular pattern; PGC-1α: Peroxisome proliferator-activated receptor-gamma coactivator -1alpha; NLR: nucleotide-binding oligomerization domain (NOD)-like receptor;T-RFLP analysis: Terminal restriction fragment length polymorphism;NOD: nucleotide-binding oligomerization domain; AMPK: adenosine 5′-monophosphate-activated protein kinase; FSTL1: follistatin-like 1; GLP: glucagon-like peptide; LIF: leukemia inhibitory factor; Metrnl: meteorin-like; TGF-β: transforming growth factor β; TNF: tumor necrosis factor; UCP1: uncoupling protein 1; GLP-1: glucagon-like peptide 1; IPost: Ischemic postconditioning; c-Met: mesenchymal-epithelial transition factor; SRF: serum response factor; NYHA: The New York Heart Association; Fas: fatty acid synthase; Acc : acetyl-CoA carboxylase; Cpt-1: Carnitine palmitoyltransferase I; β-Had: beta-Hydroxyacyl-CoA dehydrogenase; OLETF: Otsuka Long-Evans Tokushima Fatty; FATP4: fatty acid transporter4; GSK3β: glycogen synthetic kinase 3 β.
Type of Study: Review Article |
Subject:
Cellular and molecular
Received: 2025/02/17 | Accepted: 2024/12/21 | Published: 2024/12/21
Received: 2025/02/17 | Accepted: 2024/12/21 | Published: 2024/12/21
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